Ectopic Junctional Tachycardia (EJT) is a medical condition characterized by an abnormally rapid heart rhythm originating from a location within the atrioventricular (AV) junction, rather than the normal pacemaker site in the heart.
The AV junction is the region in the heart where electrical signals are transmitted between the upper chambers (atria) and the lower chambers (ventricles). In EJT, abnormal electrical activity arises in this region, resulting in a faster heart rate than usual. This fast heart rate is considered tachycardia, defined as more than 100 beats per minute.
EJT is typically caused by the presence of extra electrical pathways in the heart, which can disrupt the normal flow of electrical signals. It may also be associated with other underlying heart conditions or disease, such as congenital heart defects, inflammation of the heart, cardiac surgery, or certain medications.
The symptoms of EJT can vary from mild to severe and may include palpitations, shortness of breath, dizziness, fainting, chest discomfort, or fatigue. Diagnosis is made by conducting electrocardiogram (ECG) tests, which can identify the characteristic electrical abnormalities associated with EJT.
Treatment options for EJT aim to control the rapid heart rate and minimize symptoms. This may involve medication to slow down the heart rate, or in severe cases, electrical cardioversion or ablation procedures may be necessary to eliminate the abnormal electrical pathways. Management of any underlying heart conditions contributing to EJT is also important.
In summary, Ectopic Junctional Tachycardia refers to a rapid heart rhythm originating from an abnormal location in the heart's AV junction, requiring treatment to control the heart rate and manage associated symptoms.
